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B-cell system activation and HIV-1 infection.

Amadori A, Zamarchi R, Veronese ML, Francavilla E, Barelli A, Borri A, Chieco-Bianchi L; International Conference on AIDS.

Int Conf AIDS. 1989 Jun 4-9; 5: 640 (abstract no. Th.C.P.144).

Institute of Oncology, University of Padova, Padova, Italy

OBJECTIVE: To better define B-cell activation during HIV-1 infection. METHODS: Limiting dilution assay of B-cell precursors spontaneously secreting HIV-1-specific and total Ig; absorption of culture supernatants with solid-phase HIV-1; effect of T-cell and monocyte removal, and interleukin addition, on in vitro spontaneous Ig synthesis. RESULTS: In vitro spontaneous Ig synthesis did not depend on T-cell presence, while monocyte removal caused a striking fall in spontaneous Ig production. IL-6 presence in culture was essential; antisera against IL-6 strongly reduced spontaneous Ig synthesis, which was partly restored by rIL-6 addition to monocyte-depleted populations. The frequency of B-cell precursors spontaneously producing HIV-1-specific Ab was about 1/3-1/4 of that of spontaneously activated, Ig-secreting precursors. Moreover, after absorption with solid-phase HIV-1, about 30% of total Ig was removed from unstimulated culture supernatants. CONCLUSIONS: HIV-1-specific B-cell activation is a major constituent of the overall B-cell activation in PBL from seropositive subjects. This phenomenon can contribute to the virus-induced immune damage, and partly explain the increased frequency of AIDS-associated B-cell malignancies.

Publication Types:
  • Meeting Abstracts
Keywords:
  • AIDS Vaccines
  • Acquired Immunodeficiency Syndrome
  • B-Lymphocytes
  • HIV
  • HIV Antibodies
  • HIV Antigens
  • HIV Infections
  • HIV Seropositivity
  • HIV-1
  • Immunoglobulins
  • In Vitro
  • Interleukin-6
  • Interleukins
  • Lymphocyte Activation
  • Monocytes
  • T-Lymphocytes
  • immunology
Other ID:
  • 00339689
UI: 102179298

From Meeting Abstracts




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